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522 While hepatocyte- or intestine-specific deletion of FXR delays liver regeneration without causing mortality, global FXR KO mice exhibit severely impaired regeneration and high mortality rates

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Growth hormone is released, tissues repair, immune function consolidates, and memories are processed during these stages

Immunofluorescence double-labeling staining results demonstrated a significant increase in the proportions of F4/80-Caspase1 positive cells in the DMM group compared to the SHAM group (Fig

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